- I have finished taking Questran lite for 2 months (3 packets a day)
- I have completed a 2 month course of Bactroban (applied 3 times a day)
- I have ended the 1 month course of Doxycycline (2 times a day)
I haven’t noticed any improvement of my CFS symptoms throughout the Coagulase Negative Staphylococcus (CNS) treatment. I am a little disappointed that I didn’t improve in some way as the CNS treatment was one that I had high hopes for. This was based on my CFS onset and CNS being what I considered a likely mechanism for this onset.
I must note the difficulty I had in completing this treatment. It was almost impossible to space out the CNS treatments in order to avoid the effectiveness of certain treatments being reduced. For instance, I had Questran 3 times a day and couldn’t take any tablets 1 hour before or 7 hours after taking the Questran, as the Questran would absorb them. I also couldn’t take the probiotic within 2 hours of the Doxycycline. Adding these temporal restrictions into the mix of my normal, everyday tablet treatment made this protocol challenging to follow
I did experience some minor side effects on the CNS treatment protocol.
- After 3 weeks on the doxycycline, I experienced an outbreak of acne on my chest and back. This wasn’t just one or two isolated pimples but rather hundreds. I have never had acne on my body before hence it seems reasonable to conclude that this outbreak was most likely related to one of the CNS medications. The Doxycycline I was taking is commonly prescribed to treat severe cases of acne and hence having an acne outbreak while on this antibiotic was unexpected. A week after the acne outbreak, it has mostly cleared up. It is possible that the acne was due to the herxheimer effect.
- My rash (that I suspect is Bartonella) has slightly faded however is still present. This occurred after approximately 3 weeks on Questran. I can’t explain why this happened. One tenuous theory is the link of CNS treatment being used for Lyme Disease and my rash being Bartonella, which can be caused by Lyme Disease.
In conclusion, my lack of improvement on the modified Shoemaker protocol to treat CNS reduces but doesn’t exclude the likelihood of CNS being central to my CFS cause. I have never had an adequate test for CNS and I was using a modified treatment protocol due to living in Australia and not the US. I still find melanocyte-stimulating hormone levels interesting in regards to the Shoemaker protocol and indeed my case of CFS. MSH deficiency causes an overproduction of mucus and various mucus symptoms were a feature of my CFS onset (and are still an ongoing feature of my CFS.)